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Frequently Asked Questions What
is glaucoma? After my dog’s yearly check-up the veterinarian told me that my dog had developed glaucoma. I haven't been able to find any information on this subject. Can you help? An
abnormal elevation of the internal pressure of the eye. Glaucoma is an abnormal increase in the internal
pressure of the eye. Normally fluids continually enter and exit various
parts of the eye at a balanced rate so that the intraocular pressure
varies only slightly within a normal range. Several diseases can disrupt
this balance; a net increase of fluid builds up in the eye because the
fluid is unable to drain correctly. This causes the rise in intraocular
pressure. Measurement of intraocular pressure is a fairly simple
procedure, but it requires special instruments.
If the pressure gets high enough, irreversible
damage to the retina and optic nerve may result. This can impair vision
and may cause blindness. Substantial, acute increases in intraocular
pressure may warrant hospitalization for intravenous treatments to lower
the pressure. Chronic treatments are directed toward correcting the
underlying causes and reducing the intraocular pressure, maintaining
vision for as long as possible and delaying or preventing occurrence in
the opposite eye.
The signs a given dog or cat with glaucoma will have
depends on the underlying cause of the increased intraocular pressure.
Signs of glaucoma may include pain, squinting, spasm of the eyelid,
redness of the eye, sensitivity around the head and ocular discharge. In
certain cases of advanced glaucoma the pupil may be dilated or the eyeball
may be enlarged.
Glaucoma is classified as primary or secondary.
There is also a congenital form that manifests perinatally, but occurs
infrequently. Dogs may develop either primary or secondary forms; cats may
develop the secondary form, but primary glaucoma is an infrequent finding
in cats. Overall, cats are less likely than dogs to have glaucoma. Primary
glaucoma is most likely to affect both eyes, but the changes in
intraocular pressure may occur several months apart. Secondary glaucoma
may be unilateral or bilateral.
Primary glaucoma may result from a genetic mechanism
that produces certain structural anomalies in the eye that are responsible
for the faulty fluid drainage and the elevated intraocular pressure. Most
cases of primary glaucoma are breed-related. Cocker spaniels, springer
spaniels, poodles, beagles, basset hounds, Siberian huskies, elkhounds,
Samoyeds, Alaskan malamutes, Chows, Chinese shar peis and Afghans are
predisposed to develop this disease. Patients with primary glaucoma may be
candidates for surgical improvement of the causative anatomic deformity
after they are medically stabilized, but these procedures are rarely
completely successful or successful for long periods of time.
Secondary glaucoma develops as a consequence of some
other disease process or event. Trauma, masses, inflammation, luxation of
the lens and ocular bleeding are potential causes of secondary glaucoma.
If an underlying cause can be identified then it must be treated in
addition to the glaucoma. Glaucoma itself can be treated with topical and
oral medications, including timolol and levobunolol (topical drugs), and
methazolamide and dichlorphenamide, among other oral drugs.
If vision is lost, there are various salvage
procedures available to make the eye less painful, including removal of
the eye. If the eye is removed, a prosthetic implant ("glass
eye") can be placed in the eye socket for cosmetic purposes if
desired. Alternative procedures to removing the eye involve intraocular
surgery or pharmacological destruction of the fluid-producing tissues in
the eye. These procedures are usually performed by a veterinary
ophthalmologist. You may want to talk to your veterinarian about a
consultation with a veterinary ophthalmologist for management of your
dog’s problem.
There is a good article in the VetCentric Magazine
titled, "When a dog can't see," that you may find interesting. |
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Copyright
© 2008. Henrietta
Animal Hospital. All rights reserved.
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